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That sustained release profile is part of what makes it useful in a clinical context
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10.1038/nature25023 96
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In neurotoxin-induced mouse models of PD, KD reduces oxidative stress, rescues mitochondrial function, preserves tyrosine hydroxylase-positive neurons, and improves motor performance, reflecting improved dopaminergic neuronal survival and reduced ROS production [68, 69, 84]
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